1.
Metabolic and nutritional complications of bariatric surgery : a review.
Mesureur, L, Arvanitakis, M
Acta gastro-enterologica Belgica. 2017;(4):515-525
Abstract
Bariatric surgery is considered as the only effective durable weight-loss therapy and may be curative for obesity-related comorbidities such as diabetes. Nevertheless this surgery is not devoid of potential long-term complications such as dumping syndrome, gastroesophageal reflux disease and nutrient deficiencies. For this reason, preoperative nutritional assessment and rigorous postoperative follow-up with administration of multi-vitamins supplements and assessment of serum levels is recommended for each patient who is undergoing a bariatric surgery. The aim of this review is to identify and treat the metabolic and nutritional complications of bariatric surgery.
2.
Complications of Chronic Pancreatitis.
Ramsey, ML, Conwell, DL, Hart, PA
Digestive diseases and sciences. 2017;(7):1745-1750
-
-
Free full text
-
Abstract
Chronic pancreatitis is a disease that leads to irreversible changes in the pancreatic morphology and function. The loss of function can lead to diabetes mellitus and exocrine pancreatic insufficiency. The inflammation and fibrosis can also lead to other complications including a chronic abdominal pain syndrome, metabolic bone disease, and pancretic cancer. This article reviews our current understanding of the mechanisms and management of these complications of chronic pancreatitis.
3.
Small intestinal bacterial overgrowth: a possible risk factor for metabolic bone disease.
Anantharaju, A, Klamut, M
Nutrition reviews. 2003;(4):132-5
Abstract
Small intestinal bacterial overgrowth (SIBO) is one of the causes of malabsorption syndromes. The prevalence of metabolic bone disease in patients with SIBO is unknown, but a recent prospective case-control study indicated significant contribution of SIBO to the development of metabolic bone disease. We review this and other reports in the literature and discuss the possible mechanisms causing metabolic bone disease in patients with SIBO.
4.
[Disorders of bone metabolism after renal transplantation].
Zbroch, E, Małyszko, J, Myśliwiec, M
Przeglad lekarski. 2002;(1):43-5
Abstract
Kidney transplantation restores renal function (glomerular filtration and production of vitamin D), thereby improving renal bone disease observed during the dialysis period. But the degree of improvement is often incomplete. Some of bone disorders related to pre-transplant renal osteodystrophy can persist after transplantation. Furthermore, immunosuppressive drug regimens effect bone remodeling in kidney transplant recipients. Post-transplant bone and mineral disorders can be classified, according to their pathogenesis and appearance time, into two groups of entities: (i) bone disorders related to pre-transplant renal osteodystrophy that persist after renal transplantation and (ii) disorders arising de novo. The first group includes: secondary hyperparathyroidism, aluminium bone disease, and dialysis-related amyloid bone disease. Post-transplant bone disorders comprise a painful legs syndrome, avascular bone necrosis, and osteopenia. Glucocorticosteroids still play a significant role in the pathogenesis of bone disease in renal transplant recipients. Due to disturbances in bone remodeling that commonly occur after renal transplantation, it is important to monitor bone metabolism in these patients.